Bluegill were exposed to 3 and 30 pg/L lindane and 20 and 200 pg/L naphthalene to determine uptake rate constants, K1 depuration rate constants, K2, and bioconcentration factors, BCF. Correlations were determined between lipid normalized and non-lipid normalized BCFs, and between observed Kl, K2 and BCFs and predicted values. The K1 values for both chemicals and concentrations were similar. The K2 values were different (1.04 day~1, 0.46 day 1). Naphthalene was more rapid. BCFs for lindane (315) and naphthalene (98) were different. Lipid normalized BCFs for naphthalene were more variable than non-lipid normalized BCFs. The reverse was observed for lindane BCFs. Predicted K1, K2 , and BCFs were in agreement with observed values.

Data were collected from natural and introduced fishes present in Ferndale Lake, a small (120 ha) sport fishing reservoir in Camp County, east Texas. Levels of pH measured in the lake during the study period ranged from 3.5 to 5.3. Monthly field surveys and experimental manipulations were designed to evaluate quantitatively the signs of stress at various biological levels. Lethal limits to low pH were quantified for largemouth bass (Micropterus salmoides) and bluegill (Lepomis macrochirus) to be pH 3.8 and 4.0,respectively. Mean blood pH (+ 1 SD) of 59 bluegill was 7.41 (j 0.16), with no significant difference (P-0.05) among groups from Ferndale Lake and Moss Lake (Cooke Co., Texas) under experimental conditions, even when severe stress was externally apparent. In a dual-trough horizontal pH gradient, bluegill behavioral avoidance was observed at pH levels below 7.0. Individual testing of 40 bluegill in pH gradient of 5.2 to 7.6 resulted in median occupation of pH 7.1,with an interquartile range of pH 6.9 to 7.3. Decreased community structure and population "well being" compared to early studies cannot be attributed entirely to recent acidic condition. Separating potential stress due to lake conditions from that due to heavy biotic predation by sport fishing in …

Human chorionic gonodotropin (HCG) affected in various ways cell cultures infected with strains of Rous sarcoma virus (RSV). The cell cultures studied were chick embryo fibroblasts (CEF), normal rat kidney cells infected with temperature-sensitive mutant (LA31-NRK) and a wild type RSV (B77-NRK). HCG increased the rate of transformation and viral titer of CEF cells infected with RSV, but not B77-NRK. HCG increased significantly transformation rates of LA31-NRK, only if the temperature sensitive transformation process was first delayed by incubation at non-permissive temperatures. It is suggested that some postinfective, pretransformational event(s) may operate before viralmediated transformation rates are increased by HCG.